Category: Morphs

Go to History for more details about the DIFFUSED / BLOODRED base mutation of this compound morph.

In 1997, we acquired a Bloodred female from an unknown corn snake breeder that had a few lateral patches ofwhite.  We bred her to one or two males over the next six years, but never reproduced the white patches, nor did we hold back any of her sons to breed back to her.  In 2003, she was bred to a male Bloodred that has considerably more white on his sides, but no white was demonstrated in any of the F¹ progeny.  Upon breeding two pairs of the F¹ babies together, approximately 50% of the F²s demonstrated various degrees of white on their sides.  Also, when breeding one of the F¹males back to the SMR original female, 50% of those progeny also had various degrees of lateral white patches.  This demonstrated that the SMR P/S Bloodreds were not alleles of the original male Bloodred gene that had similar random lateral white patches. 

At this time – in my opinion – insufficient data has been gathered to determine that P/S Bloodreds owe their atypical white lateral and facial markings to a gene mutation.  It is remotely possible that polygenetic traits are responsible for the atypical patches of white on the face and sides, based on confusing phenotypes.  While evaluation of Mendelian Phenotype Proportions points to the likelihood of a gene mutation, that cannot be definitely proclaimed at this time.

It is sometimes difficult to determine the inheritance of a trait or mutation when expression of the atypical feature is highly variable – as is the case with SMR P/S Bloodreds.  In other words, are the Bloodred siblings of P/S Bloodreds that lack lateral patches of white not P/S Bloodreds OR are they P/S Bloodred mutants that are at the lowest end of the 0-to-10 scale for white expression?  When proving the mode of inheritance via evaluation of Mendelian Phenotype Proportions in a single brood of snakes, visual expression is crucial.  Hence, if the expression of white in this morph can be so extremely variable, when citing the ratio of visual mutants compared to visual non mutants, the very description of inheritance can be in question.  I therefore honestly don’t know if P/S Bloodreds owe their distinctive pied-sided white appearance to a recessive mutation OR polygenic trait modifications. Breeding trials are constantly being evaluated.  The snag in this determination is the fact that there are many siblings of the P/S Bloodreds that lack white, but have the remarkably red sides that are devoid of markings (a virtually distinct collateral trait of SMR P/S Bloodreds).  This particular trait is not foreign to the base mutation, Bloodred, but in P/S Bloodred phenotypes, expression of this shocking red trait is definitely exaggerated, compared to non P/S Bloodreds in the hobby.  Hence, the question again, “are these non P/S Bloodred siblings of those with white, P/S bloods that are exhibiting no white OR is there an associative phenotype that exaggerates the red sides, even if they don’t exhibit lateral white?”.  Some have suggested that the lack of color in places (i.e. white patches) is an extreme expression of the genetic elimination of melanin/markings.  That perhaps the genetic erradication of markings – when expressed in its’ extreme – may eliminate not only the melanin, but the other chromatophore as well?

Aside from the random lateral white feature that is obvious in most members of this morph – compared to standard Bloodreds – is the extreme diffusion – even if they don’t demonstrate any of the randomly distributed white patches on the sides.  On most – even in the absence of lateral white patches – there is an obvious line of demarcation between the dorsal and lateral pattern fields – just above the half-way point on the sides (dorso–laterally).  This stark break line between dorsal and lateral markings also begs questions about the lateral white being a mutation OR variable expression of polygenetics. 

Genetically speaking, Fluorescent corns are Amel corns that have been selectively bred to promote their target look (red or orange blotches on an orange background, with separating white blotch margins), but we have taken this to a new and better version by selectively toward stretching the markings from blotches to bands.  Since the only gene mutation they possess is Amel, the obvious distinction between Banded Fluorescents and the classic Fluorescent corn is the obvious banding.

What to expect:

The Buf corn snake was discovered by Jan Notte (corn snake breeder in Europe https://www.slangenbroed.nl/) in 2001.  It is the first corn snake gene mutation to be identified as being dominant to wild-type.  Buf corns are generally more yellow or gold than most wild-type corns and combining them with Amel (called ORANGE) they resemble Creamsicle mutants (Bufs are not hybrids).  Bufs are relatively new to the United States so expect to see many new morph compounds of this beautiful mutation in the near future.  

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INTERSPECIES  HYBRID !
Creamsicle Bloodred (aka: Diffused Creamsicle, Bloodsicles)

The HYBRID element of this morph compound was formerly considered an intergrade of what used to be two corn snake subspecies (Elaphe guttatus guttatus X Elaphe guttatus emoryi), Creamsicles are the final product of crossing an Emory’s Rat (aka: Great Plains Rat Snake) with an Amel corn. Since the new taxonomic classification assigns distinct species to each (Pantherophis emoryi and Pantherophis guttatus), in herpetocultural vernacular, Creamsicles are now officially considered hybrids.  ANY progeny from Creamsicles or any corn snake that has any degree of Emory’s Rat Snake in it, is considered a HYBRID.  The albinos are called Creamsicles and the non-albinos are often called Rootbeers.

Combination of the HYBRID Creamsicle and the Diffused mutation (see Diffused VS Bloodred history) renders this beautifully orange corn snake morph.

Glossary Term Hyperlinks:

Hypo corns have a potentially confusing name.   I refer to the reality that they were named for the Latin/Greek derived term that best describes the genetic mechanics of this mutation — Hypo (greatly reduced) and melanism (black pigment).  Hence, we use the capitalized name Hypo to apply to this particular morph and lower case hypo referencing reduction of any color or pattern in other mutations (i.e. hypoerythrism). At this time, there are at least three other hypo-type mutations in corns (i.e. Lava, Sunkissed, Ultra).

General Note:
While most of the snakes we produce will mature to resemble the featured adult image(s) on our web site, unlike manufactured products that are respectively identical to each other, the nature of polygenic variation results in no two specimens being exactly the same.  The snake we select for you may not mature to be identical to the pictured examples, but will be chosen based on our experience of observing which neonates will mature to properly represent their respective morph.  We take this responsibility very seriously, and therefore publish the guarantee that we will replace your SMR snake if it does not mature to be like our advertised examples.

Several breeders (myself included) have performed breeding trials to verify that Terrazzos are not allelic to other gene mutations.  Results were somewhat mixed – probably because people have been breeding mutations into Key Corns for so many years – but the general consensus is that most Key Corns are not allelic to current mutations.  I’ve personally bred Key Corns to Diffused (aka: bloodreds) and all the babies were phenotypes for Diffused mutants.  Many more breeding trials are in order.  Partly because of the diversity between many of the different Key habitants (including some South Dade County, Florida, corns that satisfy the visual Key Corn standard.

There are several distinct features among Lavender mutants of any variety. One baffling characteristic common in the Lavender mutation is that most of them have eye pupils that are some shade of pink – virtually identical to most albino (Amel) corn snakes.  Nobody yet knows why but some have black pupils.  Another feature in most Lavender corns is that virtually all of them hatch smaller in size than any other corn snake mutation.  What they lack in hatchling size they make up for in appetite.  Not so much that they are ravenous feeders (they actually ARE), but collectively relative to all other corn snake mutations, Lavenders statistically favor pinky mice more than any other corns we produce.  Our adult Lavender types are essentially the same size as other corn snake morphs, even though they start out so tiny. Many lavenders resemble Ghost corn snakes, as babies and adults. 

What to expect:

Go to History for more details about the DIFFUSED / BLOODRED base mutation of this compound morph.

This compound morph results from combining the color mutation, Amel with the pattern mutation Diffused.  As with most morph compounds that include the Diffused mutation, the Fire mutation‘s color affect is generally more diffused than a typical Amel corn, but nothing compared to Cayenne Fire mutants that possess the additional Red Mask mutation. Except for some of the color and pattern variants of this mutation compound, Fires arguably have much less color contrast than typical Amels and generally redder coloration. Expect dramatically reduced white margins, compared to most non-pattern Amel type mutants. 

What to expect:
Many Fire hatchlings look like little more than regular Amel Corns, but the head should have some form of Bloodred head pattern, and the belly will be devoid of classic Amel Corn snake white & off-white checkering.  The belly pattern can have colors – and even pattern – but not organized checkering.  From a short distance, adults appear to be completely red or red/orange, but upon closer examination, most have a faint blotch pattern, and some will have minute traces of white on the scales around the posterior-most dorsal markings.

Wow, what a beautiful corn snake.  Sometimes referred to as Hypo B (the second hypomelanisticmutation to be discovered), this one’s the prettiest ever.  Allegedly, the gene derives from wild-caught Okeetee corns that were caught on or near Okeetee Hunt Club.  The odd head pattern on most Sunkissed corns is very unusual for any corn snake mutation, but the older the corn snake hobby gets, the more we see such unusual things.  In some, the melanin in their pattern is sometimes “shattered”, giving the visual appearance of having dotted blotch outlines.  Some even have no discernible corn snake pattern.  Just semi-orderly dotted pattern that sometimes doesn’t even remind one of pattern saddles.  Since virtually every Sunkissed corn I’ve ever owned was less than friendly toward humans (polite way of saying, “watch out when ya reach for one“), it is safe to say that their human intolerance is a behavior associated with the mutation.

Enough of the odd characteristics of these beautiful mutants. If there is one negative stigma attached to this mutation, it’s surely the potential that the one you get may have a genetic defect sometimes called “star-gazer’s disorder”.  The is called a lethal mutation since effects of the muation are not advantageous to the homozygote.  Star-gazer’s causes the snake to have limited or aberrant control over balance.  Similar neural disorders have been demonstrated in many animal species, and sometimes the cuase is viral.  Also, the neural symptoms of this mutation parallel that of animals with certain parasites that retard balance control. Star-gazer’s in corn snakes it not a contagious disease or pathogen, so the only way your snake’s will get it is through genetics.  It is inherited recessively, so some people that swear it is not lurking in the genes of their snakes, cannot really be certain of that – without controlled breeding trials.  Only by breeding a suspect corn to a star-gazer homozygote or heterozygote can one determine the presence of the gene.  Ideally, if you have any corns that MAY have this genetic mutation, you should breed it to a known homozygote.  Even that is not proof positive, given that you must have at least 20 progeny (of which 100% are not afflicted with the disorder) in order to be reasonably assured that it’s not in your snake’s genome.  This mutation was discovered in Sunkissed mutants, but it is not linked to the Sunkissed mutation.  It has been reported in several other non-Sunkissed corns (mutant or not).  Hence, if you discover you have a star-gazer mutant, it is recommended that you restrict it’s genes to creating “control” snakes that can be used by others to determine the presence or absence of the lethal gene in their snakes.  Even though it is not transmitted like a viral pathogen, the danger of the gene inflicting many other breeding lines of snakes is likely and potentially disastrous, in the absence of breeding trials.  Such trials are under way here at SMR (and with many breeders) and if/when we determine that any of our snakes are carriers of this lethal gene, they will be euthanized.  BTW, if you think you’re safe because you have been breeding sunkissed corns (or any other corn snake type) for over four generations without seeing any homozygotes of the disorder, think again.  If your first Sunkissed corn (or Okeetee or other type) was het for this mutation, it could take many generations for you to make the discovery.  Since each snake hands one copy of its’ genotype to each of its’ progeny, potentially half of each generation could be heterozygotes.  If you (or your customers) continually bred those heterozygotes to non genetic cariers of the mutation, only part of their progeny would inherit one copy of the mutation.  If you were lucky in not seeing any sign of the gene in over four generations (or potentially unlucky, in this case), it does not follow that none of your snakes are carrying a copy of the gene.  Until you pair two of them with a copy of the gene, it will continue to hide in the family tree.  Several years ago, I bought three female Okeetees from a breeder that is now out of the corn snake trade/hobby.  They were sold as being het for Sunkissed.  I bred one of the females to one of my best Extreme Okeetees and sold the babies as Okeetees.  Two years later, a customer called me to ask why some of the Okeetee babies she produced from the pair of Okeetees she got from me were doing the loopy, corkscrew locomotive thing.  Because I had never produced a star-gazer homozygote, I naively ruled that out, but upon reviewing acquisition records, I identified that the parents of her mutants were the Okeetees het for Sunkissed.  I immediately tracked down the other two customers that had purchased some of those, advising them that those snakes could be carriers of the lethal gene.  I then euthanized the three adult female Okeetees I purchased from the other breeder.  This lethal gene could be in hundreds or thousands of corns right now, and they don’t have to be Sunkissed corns.  Hence, if you ever discover that you have the gene, advise all customers that purchased its’ progeny, and if you’re not going to use the carriers for producing TEST snakes for others, I recommend that you humanely euthanize them.  By essentially eliminating them from the gene pool, you have take an important step toward eliminating this horrible gene.

Mixing the Sunkissed mutation with other color mutations and with pattern mutants is never disappointing.  Except for the grouchy demeanor, I don’t recall seeing a single mutation compound I didn’t like.  I know you’ll have fun mixing and matching them with other corn snake mutations and morphs.